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NEUROIMAGE
Year : 2011  |  Volume : 59  |  Issue : 4  |  Page : 652-653

Pan-meningiomatosis in an operated case of cerebellopontine angle meningioma


Department of Neurosurgery, All India Institute of Medical Sciences, New Delhi, India

Date of Submission22-Apr-2011
Date of Decision24-Apr-2011
Date of Acceptance02-May-2011
Date of Web Publication30-Aug-2011

Correspondence Address:
Vivek Tandon
Department of Neurosurgery, Neurosciences Centre, AIIMS, Ansari Nagar, New Delhi
India
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Source of Support: None, Conflict of Interest: None


DOI: 10.4103/0028-3886.84369

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How to cite this article:
Phalak M, Tandon V, Mahapatra AK. Pan-meningiomatosis in an operated case of cerebellopontine angle meningioma. Neurol India 2011;59:652-3

How to cite this URL:
Phalak M, Tandon V, Mahapatra AK. Pan-meningiomatosis in an operated case of cerebellopontine angle meningioma. Neurol India [serial online] 2011 [cited 2019 Sep 23];59:652-3. Available from: http://www.neurologyindia.com/text.asp?2011/59/4/652/84369


A 51-year-old lady was operated for right cerebello-pontine angle (CP angle) meningioma by suboccipital retromastoid approach about 3.5 years back. She presented at this admission with complains of progressive difficulty in swallowing and imbalance while walking since 8 months. Her family history was not positive for NF-2 nor any other possible etiology. She had right-sided cranial nerve palsies involving trigeminal to hypoglossal nerves with long tract signs. There was no evidence of neurocutaneous markers. Contrast magnetic resonance imaging of brain revealed diffuse meningeal thickening [Figure 1]a-c. Meningeal thickening was more marked in both the CP angles and tentorium reaching up to the tentorial incisura. The patient was managed conservatively because no operative intervention was deemed suitable for such a diffuse disease.
Figure 1(a-c): Meningiomatosis in supratentorial and infratentorial compartment more marked at both CP angles and tentorial incisura

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In a study by Antinheimo et al., in the well-defined population, 1% of patients with meningioma had multiple meningiomas in association with NF2, and 4% had meningiomatosis without NF2. No families with meningiomatosis were found among the 823 patients with meningioma studied. [1] The various criteria described for NF2 clearly do not fit in our patient. [2] It is likely that contiguous growth and spread via CSF pathways may explain the diffuse meningiomatosis in our patient. [3]

 
  References Top

1.Antinheimo J, Sankila R, Carpen O, Pukkala E, Sainio M, Jaaskelainen J. Population-based analysis of sporadic and type 2 neurofibromatosis-associated meningiomas and schwannomas. Neurology 2000;54:71-6.  Back to cited text no. 1
    
2.Baser ME, Friedman JM, Wallace AJ, Ramsden RT, Joe H, Evans DG. Evaluation of clinical diagnostic criteria for neurofibromatosis 2. Neurology 2002;59:1759-65.  Back to cited text no. 2
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3.Nahser HC, Grote W, Lohr E, Gerhard L. Multiple meningiomas: Clinical and computer tomographic observations. Neuroradiology 1981;21:259-63.  Back to cited text no. 3
    


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