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ORIGINAL ARTICLE
Year : 2003  |  Volume : 51  |  Issue : 2  |  Page : 211--214

Changes in the isoprenoid pathway with transcendental meditation and Reiki healing practices in seizure disorder

RA Kumar, PA Kurup 
 Department of Neurology, Medical College Hospital, Trivandrum, Kerala, India

Correspondence Address:
P A Kurup
Gouri Sadan, T. C. 4/1525, North of Cliff House, Kattu Road, Kowdiar P.O., Trivandrum-695003, Kerala
India

A quantal perceptive model of brain function has been postulated by several groups. Reiki-like healing practices in seizure disorder (ILAE classification II E generalized seizures tonic clonic), involving transfer of life force or low level of electromagnetic force (EMF) from the healer to the recipient patient, may act via quantal perceptive mechanisms. Increased synthesis of an endogenous membrane Na+-K+ ATPase inhibitor digoxin and a related tyrosine / tryptophan transport defect has been demonstrated in refractory seizure disorder (ILAE classification II E generalized seizures tonic clonic). Reiki-like healing practices in refractory epilepsy results in a reduction in seizure frequency. Reiki-like healing practices produce membrane stabilization and stimulation of membrane Na+-K+ ATPase activity by quantal perception of low levels of EMF. The consequent intracellular hypermagnesemia inhibits HMG CoA reductase activity and digoxin synthesis resulting in the alteration of the neutral amino acid transport (tryptophan / tyrosine) defect. A hypothalamic digoxin-mediated quantal perception model of brain function is proposed. The phenomena of biological transmutation and consequent hypermagnesemia occurring in the resultant neuronal quantal state is also discussed.


How to cite this article:
Kumar R A, Kurup P A. Changes in the isoprenoid pathway with transcendental meditation and Reiki healing practices in seizure disorder .Neurol India 2003;51:211-214


How to cite this URL:
Kumar R A, Kurup P A. Changes in the isoprenoid pathway with transcendental meditation and Reiki healing practices in seizure disorder . Neurol India [serial online] 2003 [cited 2020 Sep 28 ];51:211-214
Available from: http://www.neurologyindia.com/article.asp?issn=0028-3886;year=2003;volume=51;issue=2;spage=211;epage=214;aulast=Kumar;type=0