Neurol India Close
 

Figure 1: Case 1. Magnetic resonance imaging (MRI) T1 post-gadolinium axial image showing a solid and cystic enhancing lesion in the pineal region (a; MRI). Histophotomicrographs show a tumor with papillary architecture (b and c: H and E, × 100) and epithelial cytomorphology (d and e: H and E, × 400). The tumor showed immunoreactivity for vimentin (f; IHC), p53 (i; IHC), Bcl-2 (j; IHC), S-100 protein (k; IHC, cytoplasmic), focally for cytokeratin (CK) (g; IHC) and glial fibrillary acidic protein (GFAP) (l; IHC), while it was negative for epithelial membrane antigen (EMA) (n; IHC), neuron specific enolase (NSE) (h; IHC) and synaptophysin (m) and MIB-1 LI (o; IHC) of 2-4% was noted

Figure 1: Case 1. Magnetic resonance imaging (MRI) T1 post-gadolinium axial image showing a solid and cystic enhancing lesion in the pineal region (a; MRI). Histophotomicrographs show a tumor with papillary architecture (b and c: H and E, × 100) and epithelial cytomorphology (d and e: H and E, × 400). The tumor showed immunoreactivity for vimentin (f; IHC), p53 (i; IHC), Bcl-2 (j; IHC), S-100 protein (k; IHC, cytoplasmic), focally for cytokeratin (CK) (g; IHC) and glial fibrillary acidic protein (GFAP) (l; IHC), while it was negative for epithelial membrane antigen (EMA) (n; IHC), neuron specific enolase (NSE) (h; IHC) and synaptophysin (m) and MIB-1 LI (o; IHC) of 2-4% was noted