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Figure 2: (a) Magnetic resonance imaging (MRI) diffusion weighted image (WI) axial section showing no diffusion restriction, (b) MRI susceptibility WI axial section showing no blooming, (c) T1 magnetization transfer images axial section showing hyperintense rim suggestive of caseating tuberculoma, (d-f) T1 fat-saturated post contrast shows peripheral intense enhancement with enhancing tuberculomas in the cerebellum and in left temporal pole. Leptomeningitis along cerebellar folia and in bilateral sylvian fi ssure, (g) MR perfusion showing no increased perfusion in lesion relative cerebral blood volume (rCBV-61) as compared to normal temporal white matter (rCBV-350), (h) MR spectroscopy (multi-voxel) in lesion shows prominent lipid lactate peak with choline: N-acetyl-aspartate 1:3

Figure 2: (a) Magnetic resonance imaging (MRI) diffusion weighted image (WI) axial section showing no diffusion restriction, (b) MRI susceptibility WI axial section showing no blooming, (c) T1 magnetization transfer images axial section showing hyperintense rim suggestive of caseating tuberculoma, (d-f) T1 fat-saturated post contrast shows peripheral intense enhancement with enhancing tuberculomas in the cerebellum and in left temporal pole. Leptomeningitis along cerebellar folia and in bilateral sylvian fi ssure, (g) MR perfusion showing no increased perfusion in lesion relative cerebral blood volume (rCBV-61) as compared to normal temporal white matter (rCBV-350), (h) MR spectroscopy (multi-voxel) in lesion shows prominent lipid lactate peak with choline: N-acetyl-aspartate 1:3