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Figure 1: (a-d) Magnetic resonance imaging (MRI) of the brain of the child during the acute presentation; T2 and T2 fluid-attenuated inversion recovery axial imaging of the brain showing symmetrical T2-weighted hyperintensities in the caudate, putamen (thick white arrow in a, thin black arrow in b), and posteromedial thalamus (thin white arrow in a) along with cystic changes and hyperintensities involving the cerebral cortex (thin white arrows in b and d) and cerebellum (thick black arrow in c). (e-h) MRI of the brain at 3-year follow-up after treatment revealing significant resolution of the hyperintensities in the cerebral cortex, cerebellum, and deep gray nucleus along with persistent cystic changes in the basal ganglia

Figure 1: (a-d) Magnetic resonance imaging (MRI) of the brain of the child during the acute presentation; T2 and T2 fluid-attenuated inversion recovery axial imaging of the brain showing symmetrical T2-weighted hyperintensities in the caudate, putamen (thick white arrow in a, thin black arrow in b), and posteromedial thalamus (thin white arrow in a) along with cystic changes and hyperintensities involving the cerebral cortex (thin white arrows in b and d) and cerebellum (thick black arrow in c). (e-h) MRI of the brain at 3-year follow-up after treatment revealing significant resolution of the hyperintensities in the cerebral cortex, cerebellum, and deep gray nucleus along with persistent cystic changes in the basal ganglia