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Figure 3: (a and b) Plain CT brain done 1-week postop showing an established left posterior cerebral artery territory infarct, a resection cavity in the left frontal lobe and a hemicraniectomy defect with adequately decompressed brain; (c) T2-weighted, (d and e) T1-weighted gadolinium-enhanced and (f) susceptibility-weighted (SWI) MRI sequences done 6 months later showing gliosis in the left frontal lobe with no contrast enhancement, edema or mass effect. There is a small, nodular, T2-hypointense lesion in the resection cavity with corresponding signal dropout on SWI suggestive of hemosiderin staining or calcification

Figure 3: (a and b) Plain CT brain done 1-week postop showing an established left posterior cerebral artery territory infarct, a resection cavity in the left frontal lobe and a hemicraniectomy defect with adequately decompressed brain; (c) T2-weighted, (d and e) T1-weighted gadolinium-enhanced and (f) susceptibility-weighted (SWI) MRI sequences done 6 months later showing gliosis in the left frontal lobe with no contrast enhancement, edema or mass effect. There is a small, nodular, T2-hypointense lesion in the resection cavity with corresponding signal dropout on SWI suggestive of hemosiderin staining or calcification